针对阿尔茨默氏病的重要病理过程——过多的自由基导致氧化应激及神经元变性坏死,采用自由基清除剂褪黑素限速酶N-乙酰基转移酶的真核表达载体转染胚胎干细胞,并将其诱导为神经前体细胞,移植叠氮钠所致AD小鼠记忆力、B--淀粉样蛋白、AD相关基因表达的影响以耙浦蚕赴谔迥诘姆只狝D的治疗提供新的方法和理论依据。
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数据更新时间:2023-05-31
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